[1] ERTRACHT O,MALKA A,ATAR S,et al.The mitochondria as a target for cardioprotection in acute myocardial ischemia[J].Pharmacol Ther,2014,142(1):33-40.
[2] WU N,ZHANG X,GUAN Y,et al.Hypercholesterolemia abrogates the cardioprotection of ischemic postconditioning in isolated rat heart: roles of glycogen synthase kinase-3beta and the mitochondrial permeability transition pore[J].Cell Biochem Biophys,2014,69(1):123-130.
[3] BUJA L M.The pathobiology of acute coronary syndromes:clinical implications and central role of the mitochondria[J].Tex Heart Inst J,2013,40(3):221-228.
[4] 程训民,江时森.心血管病新的治疗靶点——线粒体通透转运孔开放[J].医学研究生学报,2011,24(10):1103-1107.
[5] ZHU J,REBECCHI M J,GLASS P S,et al.Interactions of GSK-3beta with mitochondrial permeability transition pore modulators during preconditioning:age-associated differences[J].J Gerontol A Biol Sci Med Sci,2013,68(4):395-403.
[6] MIURA T,TANNO M,SATO T.Mitochondrial kinase signalling pathways in myocardial protection from ischaemia/reperfusion-induced necrosis[J].Cardiovasc Res,2010,88(1):7-15.
[7] SKYSCHALLY A,Van CASTER P,BOENGLER K,et al.Ischemic postconditioning in pigs:no causal role for RISK activation[J].Circ Res,2009,104(1):15-18.
[8] ZHONG G Q,TU R H,ZENG Z Y,et al.Novel functional role of heat shock protein 90 in protein kinase C-mediated ischemic postconditioning[J].J Surg Res,2014,189(2):198-206.
[9] 刘志刚,刘永芳,陈雪君,等,异氟烷心肌保护的分子机制的初步研究[J].东南大学学报:医学版,2011,30(5):699-702.
[10] ALDI S,TAKANO K,TOMITA K,et al.Histamine H4-receptors inhibit mast cell renin release in ischemia/reperfusion via protein kinase C epsilon-dependent aldehyde dehydrogenase type-2 activation[J].J Pharmacol Exp Ther,2014,349(3):508-517.
[11] YOGALINGAM G,HWANG S,FERREIRA J C,et al.Glyceraldehyde-3-phosphate dehydrogenase (GAPDH) phosphorylation by protein kinase Cdelta (PKCdelta) inhibits mitochondria elimination by lysosomal-like structures following ischemia and reoxygenation-induced injury[J].J Biol Chem,2013,288(26):18947-18960.
[12] TIAN Z,ZHENG H,LI J,et al.Genetically induced moderate inhibition of the proteasome in cardiomyocytes exacerbates myocardial ischemia-reperfusion injury in mice[J].Circ Res,2012,111(5):532-542.
[13] LEMOINE S,ZHU L,LEGALLOIS D,et al.Atorvastatin-induced cardioprotection of human myocardium is mediated by the inhibition of mitochondrial permeability transition pore opening via tumor necrosis factor-alpha and Janus kinase/signal transducers and activators of transcription pathway[J].Anesthesiology,2013,118(6):1373-1384.
[14] HEUSCH G,MUSIOLIK J,GEDIK N,et al.Mitochondrial STAT3 activation and cardioprotection by ischemic postconditioning in pigs with regional myocardial ischemia/reperfusion[J].Circ Res,2011,109(11):1302-1308.
[15] MEIER J A,LARNER A C.Toward a new STATe:the role of STATs in mitochondrial function[J].Semin Immunol,2014,26(1):20-28.
[16] 唐雪骁,刘光陵,夏正坤,等.人参皂甙Rg1对阿霉素诱导系膜细胞凋亡的影响[J].现代医学,2013,41(4):226-231.
[17] ROY M J,VOM A,CZABOTAR P E,et al.Cell death and the mitochondria:therapeutic targeting of the BCL-2 family-driven pathway[J].Br J Pharmacol,2014,171(8):1973-1987.
[18] DAS S,STEENBERGEN C.Mitochondrial adenine nucleotide transport and cardioprotection[J].J Mol Cell Cardiol,2012,52(2):448-453.
[19] HU S,YAN G,XU H,et al.Hypoxic preconditioning increases survival of cardiac progenitor cells via the pim-1 kinase-mediated anti-apoptotic effect[J].Circ J,2014,78(3):724-731.
[20] BORILLO G A,MASON M,QUIJADA P,et al.Pim-1 kinase protects mitochondrial integrity in cardiomyocytes[J].Circ Res,2010,106(7):1265-1274.
[21] CHEN K,LI G,GENG F,et al.Berberine reduces ischemia/reperfusion-induced myocardial apoptosis via activating AMPK and PI3K-Akt signaling in diabetic rats[J].Apoptosis,2014,19(6):946-957.
[22] MORRISON A,LI J.PPAR-gamma and AMPK-advantageous targets for myocardial ischemia/reperfusion therapy[J].Biochem Pharmacol,2011,82(3):195-200. |
