[1] YELLON DM,HAUSENLOY DJ.Myocardial reperfusion injury[J].N Engl J Med,2007,357:1121-35.
[2] MURRY CE,JENNINGS RB,REIMER KA.Preconditioning with ischemia:a delay of lethal cell injury in ischemic myocardium[J].Circulation,1986,74:1124-1136.
[3] ZHAO ZQ,CORVERA JS,HALKOS ME,et al.Inhibition of myocardial injury by ischemic postconditioning during reperfusion:comparison with ischemic preconditioning[J].Am J Physiol Heart Circ Physiol,2003,285(2):H579-H588.
[4] HAUSENLOY DJ,YELLON DM.Preconditioning and postconditioning:united at reperfusion[J].Pharmacology & Therapeutics,2007,116:173-191.
[5] VINTEN-JOHANSEN J.Postconditioning:a mechanical maneuver that triggers biological and molecular cardioprotective responses to reperfusion[J].Heart Fail Rev,2007,12:235-244.
[6] HAUSENLOY DJ,YELLON DM.Reperfusion injury salvage kinase signalling:taking a RISK for cardioprotection[J].Heart Fail Rev,2007,12:217-234.
[7] ARGAUD L,GATEAU-ROESCH O,RAISKY O,et al.Postconditioning inhibits mitochondrial permeability transition[J].Circulation,2005,111:194-197.
[8] SHANMUGANATHAN S,HAUSENLOY DJ,DUCHEN M R,et al.Mitochondrial permeability transition pore as target for cardioprotection in the human heart[J].Am J Physiol Heart Circ Physiol,2005,289:H237-H242.
[9] LIM SY,DAVIDSON SM,HAUSENLOY DJ,et al.Preconditioning and postconditioning:the essential role of the mitochondrial permeability transition pore[J].Cardiovasc Res,2007,75:530-535.
[10] COHEN MV,YANG XM,DOWNEY JM.The pH hypothesis of postconditioning staccato reperfusion reintroduces oxygen and perpetuates myocardial acidosis[J].Circulation,2007,115:1895-1903.
[11] PENNA C,RASTALDO R.Postconditioning induced cardioprotection requires signaling through a redox-sensitive mechanism,mitochondrial ATP-sensitive K⁺ channel and protein kinase C activation[J].Basic Res Cardiol,2006,101:180-189.
[12] TSURSUMI YM,YOKOYAMA T,HORIKAWA Y,et al.Reactive oxygen species trigger ischemic and pharmacological postconditioning:in vivo and in vitro characterization[J].Life Sciences,2007,81:1223-1227.
[13] PENNA C,MANCARDI D,TULLIO F,et al.Postconditioning and intermittent bradykinin induced cardioprotection require cyclooxygenase activation and prostacyclin release during reperfusion[J].Basic Res Cardiol,2008,103:368-377.
[14] JIN ZQ,KARLINER JS,VESSEY DA.Ischemic postconditioning protects isolated mouse hearts against ischemia/reperfusion injury via sphingosine kinase isoform 1 activation[J].Cardiovasc Res,2008,79(1):134-140.
[15] VESSEY DA,LI L Y,KELLEY M,et al.Sphingosine can pre-and post-condition heart and utilizes a different mechanism from sphingosine 1-phosphate[J].J Biochem Mol Toxicol,2008,22:113-118.
[16] ARGAUD L,GATEAU-ROESCH O,AUGEUL L,et al.Increased mitochondrial calcium coexists with decreased reperfusion injury in postconditioned (but not preconditioned) hearts[J].Am J Physiol Heart Circ Physiol,2008,294:H386-H391.
[17] BAXTER GF,BURLEY DS.Reperfusion and calculated RISKs:pharmacological postconditioning of human myocardium[J].Br J Pharmacol,2008,153:1-3.
[18] ZANG WJ,SUN L,YU XJ.Cardioprotection of ischemic postconditioning and pharmacological post-treatment with adenosine or acetylcholine[J].Acta Physiologica Sinica,2007,59(5):593-600.
[19] TISSIER R,WAINTRAUB X,COUVREUR N,et al.Pharmacological postconditioning with the phytoestrogen genistein[J].Journal of Molecular and Cellular Cardiology,2007,42:79-87.
[20] PENNA C,MANCARDI D,RASTALDO R,et al.Intermittent activation of bradykinin B(2) receptors and mitochondrial KATP channels trigger cardiac postconditioning through redox signaling[J].Cardiovasc Res,2007,75:168-177.
[21] JI Y,PANG QF,XU G,et al.Exogenous hydrogen sulfide postconditioning protects isolated rat hearts against ischemia-reperfusion injury[J].Eur J Pharmacol,2008,587:1-7.
[22] MUDALAGIRI NR,Di SALVO C,KOLVEKAR S,et al.Erythropoietin protects the human myocardium against hypoxia-reoxygenation injury via PI3-kinase and ERK1/2 activation[J].Br J Pharmacol,2008,153:50-56.
[23] OKA N,WANG L,MI W,et al.Cyclosporine A prevents apoptosis-related mitochondrial dysfunction after neonatal cardioplegic arrest[J].J Thorac Cardiovasc Surg,2008,135:123-30.
[24] KERENDI F,KIN H,HALKOSME,et al.Remote postconditioning brief renal ischemia and reperfusion applied before coronary artery reperfusion reduces myocardial infarct size via endogenous activation of adenosine receptors[J].Basic Res Cardiol,2005,100:404-412.
[25] ANDREKAG,VERTESALTAIM,SZANTHOG,et al.Remote ischaemic postconditioning protects the heart during acute myocardial infarction in pigs[J].Heart,2007,93:749-752.
[26] LI CM,ZHANG XH,MA XJ,et al.Limb ischemic postconditioning protects myocardium from ischemia-reperfusion injury[J].Scand Cardiovasc J,2006,40:312-317.
[27] LOUKOGEORGAKIS SP,WILLIAMS R,PANAGIOTIDOU AT,et al.Transient limb ischemia induces remote preconditioning and remote postconditioning in humans by a KATP channel-dependent mechanism[J].Circulation,2007,116:1386-1395.
[28] YANG XC,LIU Y,WANG LF,et al.Permanent reduction in myocardial infarct size by postconditioning in patients after primary coronary angioplasty[J].Circulation,2006,114:II-812.
[29] HUHN R,HEINEN A,WEBER NC,et al.Hyperglycaemia blocks sevoflurane-induced postconditioning in the rat heart in vivo:cardioprotection can be restored by blocking the mitochondrial permeability transition pore[J].Br J Anaesth,2008,100(4):465-471.
[30] MYKYTENKO J,KERENDI F,REEVES JG,et al.Long-term inhibition of myocardial infarction by postconditioning during reperfusion[J].Basic Res Cardiol,2007,102:90-100.